Methods: The sample consisted of 241 adults diagnosed with SUD. Data were collected from residential treatment programs for co-occurring SUD. The majority of the sample was non-Hispanic white (71.8%), heterosexual (83.8%), and male (58.5). Participants’ age ranged from 18-68 (mean = 33), age of drug-use onset ranged from 4-42 (mean =14.5). First drugs used were alcohol (67.5%) and marijuana (24.7%). Primary drugs of choice reported were alcohol (44.5%), heroin (33.9%), and prescribed opioids (20%). Over 80% of participants reported needing emotional help, had psychiatric admissions (32%), or attempted suicide (28%). Study measures included: Adverse Childhood Experiences scale (Felitti, et al., 1998), Self-Report Emotional Intelligence Test (Schutte, et al., 1998), Proactive Coping Scale (Greenglass, et al., 1999), Brief Resilience Scale (Smith, et al., 2008), General Self-Efficacy Scale (Schwarzer & Jerusalem, 1995), and Mental Health Screening Form III (Carroll & McGinley, 2001). Three scales were developed to measure the dependent variable SUS: meeting DSM-V diagnosis criteria, negative substance-use outcomes, and substance-use persistence despite adversity.
Results: As anticipated, hierarchical regression analysis revealed significant positive correlations between ACE (p < .05), age-of-onset (p < .05), and mental-health-pathology (p < .01), on SUS. While neither emotional intelligence nor self-efficacy maintained significance, resilience (p < .01) and coping (p < .05), were significantly negatively related to two of the three measures of SUS.
Conclusions: Despite the robust impact of ACE on SUS, coping and resilience appear to play an important buffering role, mitigating the impact of traumatic childhood experiences. Social work education and practice aimed at treating at-risk populations must integrate trauma informed practices focused on creating opportunities to develop and enhance coping skills and resilience into prevention and early intervention programs for youth, as well as, into treatment programs for clinical samples of adults already diagnosed with SUD. Future research should further explore factors that may buffer the negative impact of ACE on later SUS and the development of SUD.